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More convincing evidence should include the aggregation of lipoylated TCA cycle proteins, loss of Fe-S cluster proteins, and abnormalities in key regulators such as ferredoxin 1 (FDX1), lipoic acid synthetase (LIAS), and dihydrolipoamide S-acetyltransferase (DLAT)
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Coronavirus replication requires the formation of double-membrane vesicles (DMVs) derived from endoplasmic reticulum (ER)
or prevent any disease.
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