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Moreover, in brain of this APPM631L-modified J20 mouse, in contrast to the regular J20 mouse, Aβ42 deposition was not plaquelike but rather punctate in nature, from which we speculate that Met35 of Aβ42 conceivably could be important in oligomer and subsequent plaque formation as well as in oxidative damage associated with this neurotoxic peptide (119)

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Several suggest benefits, and the cardiac work in particular reported improved function and structure in a heart failure model (the Pharmacological Research paper reporting that NNMT inhibition improved cardiac function and structure in a heart failure model)