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Most AD cases belong to the sporadic form of AD, with a disease onset after the age of 65 (late-onset Alzheimerβs disease (LOAD)), and only approximately 5% of AD cases are caused by mutations in the genes encoding for APP or PS1/PS2 (familial Alzheimerβs disease FAD), leading to an increased production of highly amyloidogenic AΞ²42 peptides
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