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The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

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Quantity:2x 1 kg

Furthermore, the significant association between oxidative stress markers (elevated TOS and reduced TAC) and glutamine levels highlights the interplay between metabolic and oxidative pathways in ischemic stroke severity and progression