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Therefore, the loss of estrogen production may explain the more prominent Aβ neuropathology observed in women but also the switch toward an Aβ-driven prooxidant state, which underlies AD development

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An experiment docking 16 flavonoids from Lu’an melon seed tea with CutC revealed that kaempferol 3-O-rutinoside (Hua et al., 2022), quercetin 3-O-rhamnosidyl galactoside, kaempferol 3-O-rhamnosidyl galactoside, and myricetin 3-O-galactoside can bind with CutC to regulate its activity, suppress TMA production, reduce TMAO levels, and achieve anti-diabetic effects