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Thus, the increase of ATF4-mediated integrated stress response (ISR) in T315I-Bcr-Abl 32D cells remodels dTTP and GSH biosynthesis, thus avoiding DNA replication stress

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To discriminate between these two possibilities, we first diminished the reduction of thiols by knocking down GSR ( gsr-1 ) or glucose-6-phosphate dehydrogenase ( gspd-1 ), the enzyme which generates NADPH, the cofactor required by all GSSG-reducing enzymes (Fig