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The improved functioning of the BBB, enhanced clearance of metabolites and protein fragments implicated in AD from the brain - an astrocyte-dependent mechanism primarily occurring during sleep [14] - and enhanced insulin signaling, suggested to play a role in AD [33], present additional mechanisms by which melatonin may impact AD pathology
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MoMFs (CD11b hi F4/80 lnt Ly6C + ) and KCs (CD11b low F4/80 hi Ly6C β ) can be distinguished by their differential expression of cell surface markers [47]
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