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100 μL of beads resuspension was incubated with 5 μg of sucA-AMP (470 nM) and either no nucleotide, 1:1 molar ratio nucleotide (470 nM), 1:5 molar ratio nucleotide (2.35 μM), or 1:25 molar ratio nucleotide (11.75 μM)

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Furthermore, in other disease models (such as Aβ-induced nerve cell damage), highly expressed or activated VDAC1 has been clearly involved in ferroptosis, exacerbating cell death by inhibiting anti-ferroptosis pathways (such as GPX4, AMPK/mTOR, Wnt/β-catenin) (149)

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