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These data indicate that the proinflammatory cytokine, TNF-α enhances the activity of NCX1 proteins in both the Ca 2+ exit and entry mode to alter [Ca 2+ ] cyt homeostasis in airway smooth muscle cells, suggesting the involvement of the NCX1 proteins in inflammatory diseases of the human airway (White et al., 2006)
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Half a decade ago, two seminal publications shed more light on how oligodendrocytes provide metabolic support to neurons (43, 44)
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