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It is important that exogenous GSH not only significantly increased the content of total glutathione (by 40%) but also increased the endogenous content of its reduced form in cardiac tissue, thus contributing to maintenance of the redox potential of mitochondria and cardiac cells in general

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Indeed, there is a growing body of evidence that suggests that in addition to being a consequence of these chronic diseases, oxidative stress could also be a key mediator in their progression [44,45,46]