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The ergogenic benefits of phosphatidic acid supplementation appear to be independent of protein intake, suggesting that it is an integral mediator of the mechanistic/mammalian target of rapamycin (mTOR) protein complexes that control skeletal muscle protein synthesis [2, 3]
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Thus, the use of amitriptyline may serve as a βshort-cutβ past BDNF to receptor activation and have a positive impact against AD
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