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Andean populations demonstrate a typical polycythemic response marked by heightened hemoglobin levels and increased erythrocyte mass, a mechanism that enhances arterial oxygen content but imposes the pathophysiological burden of elevated blood viscosity and susceptibility to chronic mountain sickness (CMS) (65)
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H(2)O(2)-induced O(2) production by a non-phagocytic NAD(P)H oxidase causes oxidant injury
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