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From Galactorrhea to Osteopenia: Rethinking Serotonin–Prolactin Interactions | Neuropsychopharmacology
Peroxiredoxin-2 expression is increased in beta-thalassemic mouse red cells but is displaced from the membrane as a marker of oxidative stress
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Activation of the mTOR pathway, together with PI3K/Akt signaling, fosters structural plasticity that counteracts neuronal atrophy and facilitates tissue repair [89]
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