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In this study, we demonstrate that 4-HNE is generated during bacterial infection both in cell culture and in vivo , is able to penetrate the bacterial cell envelope and access the cytoplasm, and that 4-HNE generation leads to bacterial growth delay or death

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At the mechanistic level, H₂ exerts neuroprotective effects through three core mechanisms: (1) selective scavenging of toxic ROS, such as -OH and ONOO-, along with the activation of the Nrf2/ARE pathway to enhance endogenous antioxidant defenses